投稿信coverletter和highlights模板

('模板1DearProf.XXXXX,Herewithinenclosedisourpaperforconsiderationtobepublishedon“(Journalname)”.Thefurtherinformationaboutthepaperisinthefollowing:TheTitle:XXXXXXXXXXXXXXXXXXXXXXXXXXXXXXXTheAuthors:XXXXXXXXXXXXXXXXXXXXXXandXXXXXXXXXXTheauthorsclaimthatnoneofthematerialinthepaperhasbeenpublishedorisunderconsiderationforpublicationelsewhere.Iamthecorrespondingauthorandmyaddressandotherinformationisasfollows,Address:DepartmentofXXXXXXXXX,TsinghuaUniversity,Beijing,10084,E-mail:Tel:86-Fax:86-Thankyouverymuchforconsideration!SincerelyYours,Dr.XXXXXXXXXXX模板2DearXX(Editor):Hereisourpapersubmittedto"(XXJournal)".TheTitle:XXXXTheAuthors:XXXXXXandXXXInthispaper,we...Theauthorsclaimthatnoneofthematerialinthepaperhasbeenpublishedorisunderconsiderationforpublicationelsewhere.ThecorrespondingauthorisDr.XXXandhisaddressandotherinformationisasfollowing:Address:DepartmentofXXX,XXUniversity,...,Tel:+86-xxx-xxxxxxxxFax:+86-xxx-xxxxxxxxThankyouverymuchforconsideration!SincerelyYours,Dr.xxx模板3DearMr.Webelievethepapermaybeofparticularinteresttothereadersofyourjournalasit........1.Theworkdescribedhasnotbeensubmittedelsewhereforpublication,inwholeorinpart,andalltheauthorslistedhaveapprovedthemanuscriptthatisenclosed.2.IhavereadandhaveabidedbythestatementofethicalstandardsformanuscriptssubmittedtoNeuroscience.kindregards.Yoursincerely,通讯作者DearProf.Gil:Thisisamanuscriptbyandentitled“.......”.Itissubmittedtobeconsideredforpublicationasa“...”inyourjournal.Thispaperisnew.Neithertheentirepapernoranypartofitscontenthasbeenpublishedorhasbeenacceptedelsewhere.Itisnotbeingsubmittedtoanyotherjournal.Correspondenceshouldbeaddressedtoatthefollowingaddress,phoneandfaxnumber,andemailaddress:...Thanksverymuchforyourattentiontoourpaper.Sincerelyyours,投稿信模版四（实例）Case1DearEditor,Wewouldliketosubmittheenclosedmanuscriptentitled"GDNFAcutelyModulatesNeuronalExcitabilityandA-typePotassiumChannelsinMidbrainDopaminergicNeurons",whichwewishtobeconsideredforpublicationinNatureNeuroscience.GDNFhaslongbeenthoughttobeapotentneurotrophicfactorforthesurvivalofmidbraindopaminergicneurons,whicharedegeneratedinParkinson’sdisease.Inthispaper,wereportanunexpected,acuteeffectofGDNFonA-typepotassiumchannels,leadingtoapotentiationofneuronalexcitability,inthedopaminergicneuronsincultureaswellasinadultbrainslices.Further,weshowthatGDNFregulatestheK+channelsthroughamechanismthatinvolvesactivationofMAPkinase.Thus,thisstudyhasrevealed,forthefirsttime,anacutemodulationofionchannelsbyGDNF.OurfindingschallengetheclassicviewofGDNFasalong-termsurvivalfactorformidbraindopaminergicneurons,andsuggestthatthenormalfunctionofGDNFistoregulateneuronalexcitability,andconsequentlydopaminerelease.TheseresultsmayalsohaveimplicationsinthetreatmentofParkinson’sdisease.Duetoadirectcompetitionandconflictofinterest,werequestthatDrs.XXXofHarvardUniv.,andYYofYaleUniv.notbeconsideredasreviewers.Withthanksforyourconsideration,IamSincerelyyours,case2DearEditor,Wewouldliketosubmittheenclosedmanuscriptentitled"Ca2+-bindingproteinfrequeninmediatesGDNF-inducedpotentiationofCa2+channelsandtransmitterrelease",whichwewishtobeconsideredforpublicationinNeuron.WebelievethattwoaspectsofthismanuscriptwillmakeitinterestingtogeneralreadersofNeuron.First,wereportthatGDNFhasalong-termregulatoryeffectonneurotransmitterreleaseattheneuromuscularsynapses.Thisprovidesthefirstphysiologicalevidenceforaroleofthisnewfamilyofneurotrophicfactorsinfunctionalsynaptictransmission.Second,weshowthattheGDNFeffectismediatedbyenhancingtheexpressionoftheCa2+-bindingproteinfrequenin.Further,GDNFandfrequeninfacilitatesynaptictransmissionbyenhancingCa2+channelactivity,leadingtoanenhancementofCa2+influx.Thus,thisstudyhasidentified,forthefirsttime,amoleculartargetthatmediatesthelong-term,synapticactionofaneurotrophicfactor.Ourfindingsmayalsohavegeneralimplicationsinthecellbiologyofneurotransmitterrelease.HighlightsHighlightsareashortcollectionofbulletpointsthatconveythecorefindingsandprovidereaderswithaquicktextualoverviewofthearticle.Thesethreetofivebulletpointsdescribetheessenceoftheresearch.resultsorconclusions)andhighlightwhatisdistinctiveaboutit.',)